Fig. 4From: CD36/Lyn kinase interactions within macrophages promotes pulmonary fibrosis in response to oxidized phospholipidCD36 regulates accumulation macrophages after oxPL injury. CD36-null mice have less accumulation of alveolar macrophages (A) and exudate macrophages (B) 10 days after treatment with oxPL (POVPC 10 µg/gram). N = 4–12 per group. *p < 0.01 compared to WT mice treated with oxPL. C Relative size, assessed by mean fluorescent intensity of forward scatter (MFI of FSC), of alveolar macrophages. D Relative size of exudate macrophages. E Cell granularity, assessed by mean fluorescent intensity of side scatter (MFI of SSC), of alveolar macrophages. F Cell granularity of exudate macrophage. N = 4 per group. *p < 0.05 compared to WT mice treated with PBS. **p < 0.05 compared to WT mice treated with oxPLBack to article page