Fig. 5
From: Role of heparin in pulmonary cell populations in an in-vitro model of acute lung injury
hAM, hATII cells and hF apoptosis/necrosis measured by Annexin V and PI staining with flow cytometry. The proportion of live cells (Annexin V-FITC-/PI-), early apoptotic cells (Annexin V-FITC+/PI-), necrotic cells (Annexin V-FITC-/PI+), late apoptotic/necrotic cells (Annexin V-FITC+/PI+). No changes were observed in LPS treated samples or in LPS + Hep treated ones (hAM:human alveolar macrophages; hATII: human alveolar type II cells; hF: human fibroblasts; FITC: Fluorescein; PI: propidium iodide; LPS: Lipopolysaccharide from Escherichia coli 055:B5 and HEP: unfractionated heparin)