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Figure 1 | Respiratory Research

Figure 1

From: Is arginase a potential drug target in tobacco-induced pulmonary endothelial dysfunction?

Figure 1

Pulmonary endothelial function, represented as cumulative Ach dose response curves in pulmonary artery rings from smokers (n = 29) and never-smokers (n = 10). Smokers had impaired relaxation in response to Ach, when compared with never-smokers (p < 0.01; panel A). The smokers were divided into two subgroups according to the presence (ED+, n = 11) or absence (ED, n = 18) of pulmonary endothelial dysfunction (p < 0.01; panel B).

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