Fig. 3From: Tollip interaction with STAT3: a novel mechanism to regulate human airway epithelial responses to type 2 cytokinesSTAT3 inhibition reduces the excessive type 2 inflammatory response in Tollip deficient human tracheobronchial cells. STAT3 inhibition significantly reduced eotaxin-3 levels in supernatants of Tollip knockdown cells after 48 h of IL-13 treatment. P-values represent a paired student’s t-test. Data are from four healthy human donors. Each dot represents one donor. Data from one donor is the mean of three technical replicatesBack to article page