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Fig. 1 | Respiratory Research

Fig. 1

From: E-cigarette-induced pulmonary inflammation and dysregulated repair are mediated by nAChR α7 receptor: role of nAChR α7 in SARS-CoV-2 Covid-19 ACE2 receptor regulation

Fig. 1

Sub-chronic e-cig exposure augments inflammatory cell influx in BALF. Differential inflammatory cell counts were measured in BALF from mice exposed to air, PG or PG with nicotine (PG + Nic) for 30 days (2 h/day). (a) Total inflammatory cell counts were analyzed by Cellometer using AO/PI staining. Differential inflammatory cell counts were determined as percentages by flow cytometry. Absolute cell counts for (b) F4/80+ macrophages, (c) Ly6B.2+ neutrophils, (d) CD4a + T-lymphocytes, and (e) CD8a + T-lymphocytes were normalized to the total cell counts. Data are shown as mean ± SEM (n = 4–6/group; equal number of male and female mice). * P < 0.05, ** P < 0.01 between groups; & P < 0.05, compared to PG exposed WT group; # P < 0.05, compared to PG + Nic exposed WT group

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Respiratory Research

ISSN: 1465-993X

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