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Fig. 2 | Respiratory Research

Fig. 2

From: Alantolactone suppresses inflammation, apoptosis and oxidative stress in cigarette smoke-induced human bronchial epithelial cells through activation of Nrf2/HO-1 and inhibition of the NF-κB pathways

Fig. 2

CSE promoted IL-1 β, TNF-α, IL-6 and IFN-γ production in Beas-2B and NHBE cells. a CSE increased Beas-2B cell inflammatory response; b CSE increased NHBE cell inflammatory response. Beas-2B and NHBE cells were treatment with 1, 2 and 5% CSE for 24 h. Data were expressed as mean ± S.D. from three independent experiments. *P values < 0.05 compared with the control group. CSE: cigarette smoke extract

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