Fig. 13
From: Effects and molecular mechanisms of intrauterine infection/inflammation on lung development
Quantification of SP-A, SP-B and SP-C from the fetal and neonatal rat lung. Densitometric analysis revealed that pulmonary surfactant proteins including SP-A (at E17, E19, E21and P1, P3, P14), SP-B (at E19, E21and P1, P3) and SP-C (at E17, E19, E21and P1, P3, P7) were significantly decreased in intrauterine infection group compared to the control group.
*P < 0.05; ** P < 0.01, vs. control group.